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This Concept Map, created with IHMC CmapTools, has information related to: Answers_hypersensitivity, histamine release by the mast cell and the synthesis of other inflammatory mediators such as platelet-activating factor, leukotreins, bradykinins, prostaglandins, and cytokines that contribute to inflammation results in increased capillary permeability, histamine release by the mast cell and the synthesis of other inflammatory mediators such as platelet-activating factor, leukotreins, bradykinins, prostaglandins, and cytokines that contribute to inflammation results in dilation of blood vessels, The antigen/antibody complexes activate the classical complement pathway. results in activation of macrophages and cytokine production, The antigen/antibody complexes activate the classical complement pathway. results in MAC lysis of surrounding tissue, Type IV (delayed hypersensitivity) mechanisms CTLs use their TCR/CD8 to bind to peptide epitopes bound to MHC-I on infected cells or normal cells having cross-reacting epitopes and kill them through apoptosis., These antibodies bind to the surface of host cells results in opsonization of the host cells, The antigen/antibody complexes activate the classical complement pathway. results in massive inflammation, immediate hypersensivity types Type V (Stimulatory Hypersensitivity), histamine release by the mast cell and the synthesis of other inflammatory mediators such as platelet-activating factor, leukotreins, bradykinins, prostaglandins, and cytokines that contribute to inflammation results in stimulation of mucous secretion, Type IV (delayed hypersensitivity) mechanisms TH2 cells produce interleukin-4 (IL-4) and interleukin-5 (IL-5) to promote extracellular killing by eosinophils and causing tissue damage., Hypersensitivity description the immune systems cause harm to the body, constriction of bronchial airways results in wheezing and difficulty in breathing, delayed hypersensitivity type Type IV (delayed hypersensitivity), The complexes pass between the endothelial cells of blood vessels - especially those in the skin, joints, and kidneys - and become trapped on the surrounding basement membrane beneath these cells. examples serum sickness, autoimmune acute glomerulonephritis, rheumatoid arthritis, systemic lupus erythematosus, some cases of chronic viral hepatitis, the skin lesions of syphilis and leprosy, immediate hypersensivity types Type I (IgE-mediated or anaphylactic-type), Antibodies are made against a particular hormone receptor on a hormone-producing cell. results in overstimulation of those hormone-producing cells, immediate hypersensivity types Type II (antibody-dependent cytotoxicity), Type V (Stimulatory Hypersensitivity) mechanism Antibodies are made against a particular hormone receptor on a hormone-producing cell., Type IV (delayed hypersensitivity) description T8-lymphocytes become sensitized to an antigen and differentiate into cytotoxic T-lymphocytes; effector T4-lymphocytes become sensitized to an antigen and produce cytokines., Hypersensitivity categories delayed hypersensitivity
