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This Concept Map, created with IHMC CmapTools, has information related to: blanks_complement_pathways, C5b binds to the target cell and subsequently binds C6, C7, C8, and a number of monomers of C9 to form C5b6789n, the Membrane Attack Complex (MAC) benefit, finally, C5 convertase splits C5 molecules into C5a and C5b function C5a triggers inflammation, C3b binds to antigen as an opsonin benefit, C5a is a chemoattractant for phagocytes benefit, C5 convertase splits C5 molecules into C5a and C5b function C5a is a chemoattractant for phagocytes, C2 then binds to C4a and is cleaved by MASP2 into C2a and C2b. function C4b binds to antigen as an opsonin, C4b and C2a combine to form C3 convertase then C3 convertase splits C3 molecules into C3a and C3b, benefit, C3 convertase splits C3 molecules into C3a and C3b function C3a triggers inflammation, then, C3 convertase splits C3 molecules into C3a and C3b then C3b binds to the C3bBb to form C3bBb3b, a C5 convertase, C4b and C2a join to form a C3 convertase then C3 convertase splits C3 molecules into C3a and C3b, finally, The Complement Pathways type the lectin pathway, C3 convertase splits C3 molecules into C3a and C3b function C3b binds to antigen as an opsonin, activated C1s enzymatically cleaves C4 into C4a and C4b function C4a triggers inflammation, then C3 convertase splits C3 molecules into C3a and C3b, The Complement Pathways type the classical complement pathway, then C2 then binds to C4a and is cleaved by MASP2 into C2a and C2b.
